liquid scintillation counter Search Results


96
Revvity tri carb liquid scintillation counter
Tri Carb Liquid Scintillation Counter, supplied by Revvity, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Revvity liquid scintillation counter
Liquid Scintillation Counter, supplied by Revvity, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Revvity tri carb 4910 tr
Tri Carb 4910 Tr, supplied by Revvity, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Revvity liquid scintillation analyzer
PDE4 inhibition induces glycogenolysis in skeletal muscle and inhibits glucose uptake in skeletal muscle and heart. ( A ) PDE4 inhibition induces glycogenolysis in skeletal muscle. The glycogen levels in the liver and skeletal muscle (gastrocnemius) in postprandial mice are shown after treatment with the PAN-PDE4 inhibitor roflumilast (Rofl; 5 mg/kg; i.p.) or solvent control (Solv) for 90 min. Data are expressed as mg of glycogen per g of tissue weight. ( B ) PAN-PDE4 inhibition reduces locomotion, as reflected by reduced travel distance. Mice were injected with the PDE4 inhibitor rolipram (3 mg/kg; i.p.; n = 6) or solvent control, placed immediately in a new cage, and then locomotion was assessed using SmartCageTM technology. Traces represent changes in travel distance (cm per 5 min interval). ( C ) Postprandial mice were injected with the PDE4 inhibitor roflumilast (5 mg/kg; i.p.) or solvent control, followed 15 min later by injection with [ 3 H]-2-deoxyglucose (intravenous, i.v.). Animals were euthanized 30 min after the tracer injection, their tissues were homogenized, and the phosphorylated [ 3 H]-2-deoxyglucose-6-phosphate was isolated by anion exchange chromatography and quantified by <t>scintillation</t> counting. Data are expressed as pmol [ 3 H]-2-deoxyglucose-6-phosphate per g of tissue wet weight. All data represent the mean ± SEM. Statistical significance was determined using the Mann–Whitney test (bar graphs) or a two-way ANOVA with Sidak’s post hoc test (time courses) and is indicated as ns ( p > 0.05), * ( p < 0.05), and ** ( p < 0.01).
Liquid Scintillation Analyzer, supplied by Revvity, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/liquid+scintillation+counter/TRI-CARB+4810TR+Liquid+Scintillation+Counter/pmc09963939-202-40-43
Average 91 stars, based on 1 article reviews
liquid scintillation analyzer - by Bioz Stars, 2026-09
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EG&G Technical Services wallac 1414 winspectral2 liquid scintillation counter
PDE4 inhibition induces glycogenolysis in skeletal muscle and inhibits glucose uptake in skeletal muscle and heart. ( A ) PDE4 inhibition induces glycogenolysis in skeletal muscle. The glycogen levels in the liver and skeletal muscle (gastrocnemius) in postprandial mice are shown after treatment with the PAN-PDE4 inhibitor roflumilast (Rofl; 5 mg/kg; i.p.) or solvent control (Solv) for 90 min. Data are expressed as mg of glycogen per g of tissue weight. ( B ) PAN-PDE4 inhibition reduces locomotion, as reflected by reduced travel distance. Mice were injected with the PDE4 inhibitor rolipram (3 mg/kg; i.p.; n = 6) or solvent control, placed immediately in a new cage, and then locomotion was assessed using SmartCageTM technology. Traces represent changes in travel distance (cm per 5 min interval). ( C ) Postprandial mice were injected with the PDE4 inhibitor roflumilast (5 mg/kg; i.p.) or solvent control, followed 15 min later by injection with [ 3 H]-2-deoxyglucose (intravenous, i.v.). Animals were euthanized 30 min after the tracer injection, their tissues were homogenized, and the phosphorylated [ 3 H]-2-deoxyglucose-6-phosphate was isolated by anion exchange chromatography and quantified by <t>scintillation</t> counting. Data are expressed as pmol [ 3 H]-2-deoxyglucose-6-phosphate per g of tissue wet weight. All data represent the mean ± SEM. Statistical significance was determined using the Mann–Whitney test (bar graphs) or a two-way ANOVA with Sidak’s post hoc test (time courses) and is indicated as ns ( p > 0.05), * ( p < 0.05), and ** ( p < 0.01).
Wallac 1414 Winspectral2 Liquid Scintillation Counter, supplied by EG&G Technical Services, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
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ALOKA Co Ltd liquid scintillation counter lsc-5100
PDE4 inhibition induces glycogenolysis in skeletal muscle and inhibits glucose uptake in skeletal muscle and heart. ( A ) PDE4 inhibition induces glycogenolysis in skeletal muscle. The glycogen levels in the liver and skeletal muscle (gastrocnemius) in postprandial mice are shown after treatment with the PAN-PDE4 inhibitor roflumilast (Rofl; 5 mg/kg; i.p.) or solvent control (Solv) for 90 min. Data are expressed as mg of glycogen per g of tissue weight. ( B ) PAN-PDE4 inhibition reduces locomotion, as reflected by reduced travel distance. Mice were injected with the PDE4 inhibitor rolipram (3 mg/kg; i.p.; n = 6) or solvent control, placed immediately in a new cage, and then locomotion was assessed using SmartCageTM technology. Traces represent changes in travel distance (cm per 5 min interval). ( C ) Postprandial mice were injected with the PDE4 inhibitor roflumilast (5 mg/kg; i.p.) or solvent control, followed 15 min later by injection with [ 3 H]-2-deoxyglucose (intravenous, i.v.). Animals were euthanized 30 min after the tracer injection, their tissues were homogenized, and the phosphorylated [ 3 H]-2-deoxyglucose-6-phosphate was isolated by anion exchange chromatography and quantified by <t>scintillation</t> counting. Data are expressed as pmol [ 3 H]-2-deoxyglucose-6-phosphate per g of tissue wet weight. All data represent the mean ± SEM. Statistical significance was determined using the Mann–Whitney test (bar graphs) or a two-way ANOVA with Sidak’s post hoc test (time courses) and is indicated as ns ( p > 0.05), * ( p < 0.05), and ** ( p < 0.01).
Liquid Scintillation Counter Lsc 5100, supplied by ALOKA Co Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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90
EG&G Technical Services microbeta jet scintillation counter
PDE4 inhibition induces glycogenolysis in skeletal muscle and inhibits glucose uptake in skeletal muscle and heart. ( A ) PDE4 inhibition induces glycogenolysis in skeletal muscle. The glycogen levels in the liver and skeletal muscle (gastrocnemius) in postprandial mice are shown after treatment with the PAN-PDE4 inhibitor roflumilast (Rofl; 5 mg/kg; i.p.) or solvent control (Solv) for 90 min. Data are expressed as mg of glycogen per g of tissue weight. ( B ) PAN-PDE4 inhibition reduces locomotion, as reflected by reduced travel distance. Mice were injected with the PDE4 inhibitor rolipram (3 mg/kg; i.p.; n = 6) or solvent control, placed immediately in a new cage, and then locomotion was assessed using SmartCageTM technology. Traces represent changes in travel distance (cm per 5 min interval). ( C ) Postprandial mice were injected with the PDE4 inhibitor roflumilast (5 mg/kg; i.p.) or solvent control, followed 15 min later by injection with [ 3 H]-2-deoxyglucose (intravenous, i.v.). Animals were euthanized 30 min after the tracer injection, their tissues were homogenized, and the phosphorylated [ 3 H]-2-deoxyglucose-6-phosphate was isolated by anion exchange chromatography and quantified by <t>scintillation</t> counting. Data are expressed as pmol [ 3 H]-2-deoxyglucose-6-phosphate per g of tissue wet weight. All data represent the mean ± SEM. Statistical significance was determined using the Mann–Whitney test (bar graphs) or a two-way ANOVA with Sidak’s post hoc test (time courses) and is indicated as ns ( p > 0.05), * ( p < 0.05), and ** ( p < 0.01).
Microbeta Jet Scintillation Counter, supplied by EG&G Technical Services, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
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90
Wallac Oy liquid scintillation counter wallac 1409
PDE4 inhibition induces glycogenolysis in skeletal muscle and inhibits glucose uptake in skeletal muscle and heart. ( A ) PDE4 inhibition induces glycogenolysis in skeletal muscle. The glycogen levels in the liver and skeletal muscle (gastrocnemius) in postprandial mice are shown after treatment with the PAN-PDE4 inhibitor roflumilast (Rofl; 5 mg/kg; i.p.) or solvent control (Solv) for 90 min. Data are expressed as mg of glycogen per g of tissue weight. ( B ) PAN-PDE4 inhibition reduces locomotion, as reflected by reduced travel distance. Mice were injected with the PDE4 inhibitor rolipram (3 mg/kg; i.p.; n = 6) or solvent control, placed immediately in a new cage, and then locomotion was assessed using SmartCageTM technology. Traces represent changes in travel distance (cm per 5 min interval). ( C ) Postprandial mice were injected with the PDE4 inhibitor roflumilast (5 mg/kg; i.p.) or solvent control, followed 15 min later by injection with [ 3 H]-2-deoxyglucose (intravenous, i.v.). Animals were euthanized 30 min after the tracer injection, their tissues were homogenized, and the phosphorylated [ 3 H]-2-deoxyglucose-6-phosphate was isolated by anion exchange chromatography and quantified by <t>scintillation</t> counting. Data are expressed as pmol [ 3 H]-2-deoxyglucose-6-phosphate per g of tissue wet weight. All data represent the mean ± SEM. Statistical significance was determined using the Mann–Whitney test (bar graphs) or a two-way ANOVA with Sidak’s post hoc test (time courses) and is indicated as ns ( p > 0.05), * ( p < 0.05), and ** ( p < 0.01).
Liquid Scintillation Counter Wallac 1409, supplied by Wallac Oy, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Wallac Oy 1450 microbeta trilux liquid scintillation and luminescence counter
PDE4 inhibition induces glycogenolysis in skeletal muscle and inhibits glucose uptake in skeletal muscle and heart. ( A ) PDE4 inhibition induces glycogenolysis in skeletal muscle. The glycogen levels in the liver and skeletal muscle (gastrocnemius) in postprandial mice are shown after treatment with the PAN-PDE4 inhibitor roflumilast (Rofl; 5 mg/kg; i.p.) or solvent control (Solv) for 90 min. Data are expressed as mg of glycogen per g of tissue weight. ( B ) PAN-PDE4 inhibition reduces locomotion, as reflected by reduced travel distance. Mice were injected with the PDE4 inhibitor rolipram (3 mg/kg; i.p.; n = 6) or solvent control, placed immediately in a new cage, and then locomotion was assessed using SmartCageTM technology. Traces represent changes in travel distance (cm per 5 min interval). ( C ) Postprandial mice were injected with the PDE4 inhibitor roflumilast (5 mg/kg; i.p.) or solvent control, followed 15 min later by injection with [ 3 H]-2-deoxyglucose (intravenous, i.v.). Animals were euthanized 30 min after the tracer injection, their tissues were homogenized, and the phosphorylated [ 3 H]-2-deoxyglucose-6-phosphate was isolated by anion exchange chromatography and quantified by <t>scintillation</t> counting. Data are expressed as pmol [ 3 H]-2-deoxyglucose-6-phosphate per g of tissue wet weight. All data represent the mean ± SEM. Statistical significance was determined using the Mann–Whitney test (bar graphs) or a two-way ANOVA with Sidak’s post hoc test (time courses) and is indicated as ns ( p > 0.05), * ( p < 0.05), and ** ( p < 0.01).
1450 Microbeta Trilux Liquid Scintillation And Luminescence Counter, supplied by Wallac Oy, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Wallac Oy liquid scintillation counter wallac 1414
PDE4 inhibition induces glycogenolysis in skeletal muscle and inhibits glucose uptake in skeletal muscle and heart. ( A ) PDE4 inhibition induces glycogenolysis in skeletal muscle. The glycogen levels in the liver and skeletal muscle (gastrocnemius) in postprandial mice are shown after treatment with the PAN-PDE4 inhibitor roflumilast (Rofl; 5 mg/kg; i.p.) or solvent control (Solv) for 90 min. Data are expressed as mg of glycogen per g of tissue weight. ( B ) PAN-PDE4 inhibition reduces locomotion, as reflected by reduced travel distance. Mice were injected with the PDE4 inhibitor rolipram (3 mg/kg; i.p.; n = 6) or solvent control, placed immediately in a new cage, and then locomotion was assessed using SmartCageTM technology. Traces represent changes in travel distance (cm per 5 min interval). ( C ) Postprandial mice were injected with the PDE4 inhibitor roflumilast (5 mg/kg; i.p.) or solvent control, followed 15 min later by injection with [ 3 H]-2-deoxyglucose (intravenous, i.v.). Animals were euthanized 30 min after the tracer injection, their tissues were homogenized, and the phosphorylated [ 3 H]-2-deoxyglucose-6-phosphate was isolated by anion exchange chromatography and quantified by <t>scintillation</t> counting. Data are expressed as pmol [ 3 H]-2-deoxyglucose-6-phosphate per g of tissue wet weight. All data represent the mean ± SEM. Statistical significance was determined using the Mann–Whitney test (bar graphs) or a two-way ANOVA with Sidak’s post hoc test (time courses) and is indicated as ns ( p > 0.05), * ( p < 0.05), and ** ( p < 0.01).
Liquid Scintillation Counter Wallac 1414, supplied by Wallac Oy, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ALOKA Co Ltd liquid scintillation counter
PDE4 inhibition induces glycogenolysis in skeletal muscle and inhibits glucose uptake in skeletal muscle and heart. ( A ) PDE4 inhibition induces glycogenolysis in skeletal muscle. The glycogen levels in the liver and skeletal muscle (gastrocnemius) in postprandial mice are shown after treatment with the PAN-PDE4 inhibitor roflumilast (Rofl; 5 mg/kg; i.p.) or solvent control (Solv) for 90 min. Data are expressed as mg of glycogen per g of tissue weight. ( B ) PAN-PDE4 inhibition reduces locomotion, as reflected by reduced travel distance. Mice were injected with the PDE4 inhibitor rolipram (3 mg/kg; i.p.; n = 6) or solvent control, placed immediately in a new cage, and then locomotion was assessed using SmartCageTM technology. Traces represent changes in travel distance (cm per 5 min interval). ( C ) Postprandial mice were injected with the PDE4 inhibitor roflumilast (5 mg/kg; i.p.) or solvent control, followed 15 min later by injection with [ 3 H]-2-deoxyglucose (intravenous, i.v.). Animals were euthanized 30 min after the tracer injection, their tissues were homogenized, and the phosphorylated [ 3 H]-2-deoxyglucose-6-phosphate was isolated by anion exchange chromatography and quantified by <t>scintillation</t> counting. Data are expressed as pmol [ 3 H]-2-deoxyglucose-6-phosphate per g of tissue wet weight. All data represent the mean ± SEM. Statistical significance was determined using the Mann–Whitney test (bar graphs) or a two-way ANOVA with Sidak’s post hoc test (time courses) and is indicated as ns ( p > 0.05), * ( p < 0.05), and ** ( p < 0.01).
Liquid Scintillation Counter, supplied by ALOKA Co Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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liquid scintillation counter - by Bioz Stars, 2026-09
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PDE4 inhibition induces glycogenolysis in skeletal muscle and inhibits glucose uptake in skeletal muscle and heart. ( A ) PDE4 inhibition induces glycogenolysis in skeletal muscle. The glycogen levels in the liver and skeletal muscle (gastrocnemius) in postprandial mice are shown after treatment with the PAN-PDE4 inhibitor roflumilast (Rofl; 5 mg/kg; i.p.) or solvent control (Solv) for 90 min. Data are expressed as mg of glycogen per g of tissue weight. ( B ) PAN-PDE4 inhibition reduces locomotion, as reflected by reduced travel distance. Mice were injected with the PDE4 inhibitor rolipram (3 mg/kg; i.p.; n = 6) or solvent control, placed immediately in a new cage, and then locomotion was assessed using SmartCageTM technology. Traces represent changes in travel distance (cm per 5 min interval). ( C ) Postprandial mice were injected with the PDE4 inhibitor roflumilast (5 mg/kg; i.p.) or solvent control, followed 15 min later by injection with [ 3 H]-2-deoxyglucose (intravenous, i.v.). Animals were euthanized 30 min after the tracer injection, their tissues were homogenized, and the phosphorylated [ 3 H]-2-deoxyglucose-6-phosphate was isolated by anion exchange chromatography and quantified by scintillation counting. Data are expressed as pmol [ 3 H]-2-deoxyglucose-6-phosphate per g of tissue wet weight. All data represent the mean ± SEM. Statistical significance was determined using the Mann–Whitney test (bar graphs) or a two-way ANOVA with Sidak’s post hoc test (time courses) and is indicated as ns ( p > 0.05), * ( p < 0.05), and ** ( p < 0.01).

Journal: International Journal of Molecular Sciences

Article Title: Acute PDE4 Inhibition Induces a Transient Increase in Blood Glucose in Mice

doi: 10.3390/ijms24043260

Figure Lengend Snippet: PDE4 inhibition induces glycogenolysis in skeletal muscle and inhibits glucose uptake in skeletal muscle and heart. ( A ) PDE4 inhibition induces glycogenolysis in skeletal muscle. The glycogen levels in the liver and skeletal muscle (gastrocnemius) in postprandial mice are shown after treatment with the PAN-PDE4 inhibitor roflumilast (Rofl; 5 mg/kg; i.p.) or solvent control (Solv) for 90 min. Data are expressed as mg of glycogen per g of tissue weight. ( B ) PAN-PDE4 inhibition reduces locomotion, as reflected by reduced travel distance. Mice were injected with the PDE4 inhibitor rolipram (3 mg/kg; i.p.; n = 6) or solvent control, placed immediately in a new cage, and then locomotion was assessed using SmartCageTM technology. Traces represent changes in travel distance (cm per 5 min interval). ( C ) Postprandial mice were injected with the PDE4 inhibitor roflumilast (5 mg/kg; i.p.) or solvent control, followed 15 min later by injection with [ 3 H]-2-deoxyglucose (intravenous, i.v.). Animals were euthanized 30 min after the tracer injection, their tissues were homogenized, and the phosphorylated [ 3 H]-2-deoxyglucose-6-phosphate was isolated by anion exchange chromatography and quantified by scintillation counting. Data are expressed as pmol [ 3 H]-2-deoxyglucose-6-phosphate per g of tissue wet weight. All data represent the mean ± SEM. Statistical significance was determined using the Mann–Whitney test (bar graphs) or a two-way ANOVA with Sidak’s post hoc test (time courses) and is indicated as ns ( p > 0.05), * ( p < 0.05), and ** ( p < 0.01).

Article Snippet: After washing the columns twice with 1 mL of deionized water, the [ 3 H]-2-deoxyglucose-6-phosphate was eluted from the columns using 2 × 1 ml of 1.5 M NaCl, and the radioactivity in the eluates was then quantified using a liquid scintillation analyzer (PerkinElmer Tri-Carb ® 4810 TR).

Techniques: Inhibition, Solvent, Control, Injection, Isolation, Chromatography, MANN-WHITNEY